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No medical or therapeutic claims are made.

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These results were in accordance with other results published previously by the present groups, where the AEDG peptide and the mixture of AEDG, KED, KED and AED peptides increased expression GAP43 and Nestin genes and proteins in hPDLSCs [27]

This locally produced EPO has been shown in diverse model systems to antagonize the production and effects of proinflammatory molecules, for example, tumor necrosis factor (TNF) (4), as well as activate healing processes

First identified in 1973 by researcher Loren Pickart, GHK-Cu emerged from observations that blood plasma from younger individuals could cause older liver tissue to synthesize proteins in patterns characteristic of younger tissue